Molecular Mechanism of Congenital Heart Disease and Pulmonary Hypertension

Lieferzeit: Lieferbar innerhalb 14 Tagen

53,49 

ISBN: 981151187X
ISBN 13: 9789811511875
Herausgeber: Toshio Nakanishi/H Scott Baldwin/Jeffrey R Fineman et al
Verlag: Springer Verlag GmbH
Umfang: xiii, 405 S., 14 s/w Illustr., 74 farbige Illustr., 74 farbige Tab., 405 p. 88 illus., 74 illus. in color.
Erscheinungsdatum: 11.09.2020
Auflage: 1/2020
Produktform: Kartoniert
Einband: KT

This open access book focuses on the molecular mechanism of congenital heart disease and pulmonary hypertension, offering new insights into the development of pulmonary circulation and the ductus arteriosus. It describes in detail the molecular mechanisms involved in the development and morphogenesis of the heart, lungs and ductus arteriosus, covering a range of topics such as gene functions, growth factors, transcription factors and cellular interactions, as well as stem cell engineering technologies. The book also presents recent advances in our understanding of the molecular mechanism of lung development, pulmonary hypertension and molecular regulation of the ductus arteriosus. As such, it is an ideal resource for physicians, scientists and investigators interested in the latest findings on the origins of congenital heart disease and potential future therapies involving pulmonary circulation/hypertension and the ductus arteriosus.

Artikelnummer: 9865610 Kategorie:

Beschreibung

Autorenporträt

Toshio Nakanishi Tokyo Women's Medical University Department of Pediatric Cardiology Tokyo, Japan nakanishi.toshio@twmu.ac.jp Scott Baldwin Vanderbilt University Medical Center Department of Pediatrics (Cardiology) and Cell and Development Biology Nashville Tennessee, USA scott.baldwin@Vanderbilt.Edu Jeffrey Fineman University of California UCSF Benioff Children's Hospital San Francisco California, USA Jeff.Fineman@ucsf.edu Hiroyuki Yamagishi Keio University School of Medicine Department of Pediatrics Shinjyukuku Tokyo, Japan hyamag@keio.jp

Das könnte Ihnen auch gefallen …