Cerebral Vasospasm: Neurovascular Events After Subarachnoid Hemorrhage

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213,99 

Acta Neurochirurgica Supplement 115

ISBN: 3709117399
ISBN 13: 9783709117392
Herausgeber: Mario Zuccarello/Joseph F Clark/Gail Pyne-Geithman et al
Verlag: Springer Verlag GmbH
Umfang: xiv, 338 S.
Erscheinungsdatum: 20.09.2014
Auflage: 1/2014
Produktform: Kartoniert
Einband: Kartoniert

InhaltsangabeCLINICAL TRIALS AND DILEMMAS: History and definition of delayed cerebral ischemia. – Clinical, transcranial Doppler ultrasound, radiological features and prognostic significance of delayed cerebral ischemia. – Identifying patient report outcomes relevant to aneurysmal subarachnoid hemorrhage follow-up. – Global cerebral atrophy after subarachnoid hemorrhage: A possible marker of acute brain injury and assessment of its impact on outcome. – Protein biomarkers in patients with subarachnoid hemorrhage, vasospasm and delayed ischemic neurological deficits. – Randomized trial of clazosentan, an endothelin receptor antagonist, in patients with aneurysmal subarachnoid hemorrhage undergoing surgical clipping (CONSCIOUS-2). – Angiographic vasospasm versus cerebral infarction as outcome measures after aneurysmal subarachnoid hemorrhage. – Development of Nicardipine prolonged-release implants after clipping for preventing cerebral vasospasm: From laboratory to clinical trial. – Magnesium sulphate for aneurysmal subarachnoid hemorrhage: Why, how, and current controversy. – Prolonged intravenous infusion of sodium nitrite delivers nitric oxide (NO) in healthy volunteers. – Special article: Proposed mechanism of cerebral vasospasm: Our hypothesis and current topics. – NEUROCRITICAL CARE: Monitoring of cerebral hemodynamics and oxygenation to detect delayed ischemic neurological deficit after aneurysmal subarachnoid hemorrhage: – Usefulness of three dimensional computed tomography to quantify the subarachnoid hemorrhage volume: Prediction of symptomatic vasospasm. – COSBID-M3: A platform for multimodal monitoring, data collection and research in neurocritical care. -Transcranial Doppler as a routine in the treatment of vasospasm following  subarachnoid hemorrhage. – Systemic interleukin-6 levels reflect illness course and prognosis of patients with spontaneous nonanueyrsmal subarachnoid hemorrhage. – Beneficial effect of selective intra-arterial infusion of fasudil hydrochloride as a treatment of  symptomatic vasospasm following subarachnoid hemorrhage. – SURGICAL AND ENDOVASCULAR MANAGEMENT: Removal of clots in subarachnoid space could reduce the vasospasm after subarachnoid hemorrhage. – Effect of lumbar puncture in patients with neurismal subarachnoid hemorrhage treated microsurgically or endovascularly. – Effect of aneurysm treatment modalities on cerebral vasospasm after neurismal subarachnoid hemorrhage. – Endovascular management of post-hemorrhagic cerebral vasospasm: Indications, technical nuances and results. – The concept of a hybrid operation room: Applications in cerebrovascular surgery. – Computerized occlusion rating of embolized ruptured intracranial aneurysms: Relation to intra- and postinterventional aneurysm rehemorrhage. – SPREADING DEPOLARIZATIONS: Spreading ischemia after aneurismal subarachnoid hemorrhage. – Full-band electrocorticography of spreading depolarizations in patients with neurismal subarachnoid hemorrhage. – Cerebral glucose and spreading depolarization in patients with neurismal subarachnoid hemorrhage. – Evaluation of intracranial electrocorticography recording strips and tissue partial pressure of oxygen and temperature probes for radiofrequency induced heating. – Criteria for the diagnosis of non-infectious and infectious complications after neurismal subarachnoid hemorrhage. – Neurovascular coupling during spreading depolarizations. – NEUROVASCULAR COUPLING AND MICROVASCULATURE: Subarachnoid blood converts neurally evoked vasodilation to vasoconstriction in rat brain cortex. – Impact of subarachnoid hemorrhage on parenchymal arteriolar function. – SAH-induced suppression of voltage-gated K+ (Kv) channel currents in parenchymal arteriolar myocytes involves activation of the HB-EGF/EGFR pathway. – Mechanisms of microthrombosis and microcirculatory constriction after experimental subarachnoid hemorrhage. – Cerebral hemodynamic and metaboli

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